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Ransferring into the use of unleaded gasoline, this study employed the two types of gasoline currently used in Egypt and focused on comparing their impact. The study was designed to evaluate the effects of gasoline vapour inhalation on the brain monoamine neurotransmitters, lipid peroxidation, GSH, SOD, Na+, K+-ATPase and AChE. In addition to these biochemical analyses, the impact of gasoline inha
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Zoic acid (DTNB) which is then read at 412 nm. Determination of lipid peroxidation product (malondialdehyde, MDA) in brain tissue homogenate This method is based on the determination of malondialdehyde (MDA), an end product of lipid peroxidation, which reacts with thiobarbituric acid to yield a pink coloured trimethine complex exhibiting a maximum absorbance at 530-532 nm [28]. HPLC determination
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1/wjv.v4.i3.LIVE ATTENUATED AND INACTIVATED, RECOMBINANT SUBUNIT VACCINESCurrently, the1/wjv.v4.i3.LIVE ATTENUATED AND INACTIVATED, RECOMBINANT SUBUNIT VACCINESCurrently, the majority of vaccines licensed for human uses include live-attenuated and inactivated or killed [1] vaccines . They came from disease-causing viruses or bacteria manipulated in vitro to reduce or attenuate the pathogenic
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Ving pathogens , these types of vaccines are usually safe compared toVing pathogens , these types of vaccines are usually safe compared to live attenuated vaccines. Overall, these technologies have allowed to achieve the successes of vaccinology in the last century and to produce the vaccine formulations available on the market. However many new vaccines are needed and for them , [8] new str
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S by enzymes, e.g., enamelysin (matrix metalloproteinase 20, Mmp-20) and kallikrein 4 [49]. The ways in which these proteins guide the formation of HA in enamel is unknown. There are, however, some important clues from recent studies. In the teeth of mice that produce a truncated form of ameloblastin, enamel fails to appear [50,51]. These mice also have reduced expression of amelogenin with no app
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Tion, and additional tumor initiating pathways promote mammary carcinogenesis. We have previously shown that activated ERK1/2 levels and the migratory action of TERT-siSFRP1 cells are drastically reduced in response to TGF-R inhibition which is consistent with work described by Imamichi et al. showing that TGF- signaling mediates the cellular migration of breast cancer cells by ERK1/2 activation [
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Ded many of the proteins that had been found to be decreased under baseline conditions in uninfected SP-A-/- mice as compared to WT mice. Our working hypotheses to explain these differences are: a) that expression of these proteins is stimulated by the induction of infection, probably as a consequence of increased levels of regulatory molecules such as cytokines; b) in order to compensate for the
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Er, a barometer, thermometer and hygrometer were installed in order to continuously register the pressure, temperature and humidity, respectively. The animals were kept in an inhalation chamber and the gasoline was evaporated in a vaporizer and the vapours passed through a rubber tube to the inhalation chamber. By calculating the amount of gasoline vapours was determined as part per million (ppm)